重组小鼠Macrophage Inflammatory蛋白1 alpha / CCL3 (ab134632)
Key features and details
- Expression system: Escherichia coli
- Purity: > 90% SDS-PAGE
- Tags: His tag N-Terminus
- Suitable for: MS, SDS-PAGE
描述
-
产品名称
重组小鼠Macrophage Inflammatory蛋白1 alpha / CCL3
参阅全部 MIP-1 alpha/CCL3 蛋白酶 -
纯度
> 90 % SDS-PAGE.
ab134632 was purified using conventional chromatography techniques. -
表达系统
Escherichia coli -
Accession
-
蛋白长度
Full length protein -
无动物成分
No -
性质
Recombinant -
-
种属
Mouse -
序列
MGSSHHHHHH SSGLVPRGSH MGSMAPYGAD TPTACCFSYS RKIPRQFIVD YFETSSLCSQ PGVIFLTKRN RQICADSKET WVQEYITDLE LNA -
预测分子量
10 kDa including tags -
氨基酸
24 to 92 -
标签
His tag N-Terminus
-
-
描述
重组小鼠MIP-1 alpha/CCL3蛋白
相关产品
-
Related Products
技术指标
Our Abpromise guarantee covers the use of ab134632 in the following tested applications.
The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.
-
应用
Mass Spectrometry
SDS-PAGE
-
质谱法
MALDI-TOF -
形式
Liquid -
Concentration information loading...
制备和贮存
-
稳定性和存储
Shipped at 4°C. Store at +4°C short term (1-2 weeks). Upon delivery aliquot. Store at -20°C or -80°C. Avoid freeze / thaw cycle.
pH: 3.50
Constituents: 0.53% Sodium citrate, 10% Glycerol (glycerin, glycerine)
常规信息
-
别名
- C C motif chemokine 3
- CCL 3
- CCL3
see all -
功能
Monokine with inflammatory and chemokinetic properties. Binds to CCR1, CCR4 and CCR5. One of the major HIV-suppressive factors produced by CD8+ T-cells. Recombinant MIP-1-alpha induces a dose-dependent inhibition of different strains of HIV-1, HIV-2, and simian immunodeficiency virus (SIV). -
序列相似性
Belongs to the intercrine beta (chemokine CC) family. -
翻译后修饰
N-terminal processed form LD78-alpha(4-69) is produced by proteolytic cleavage after secretion from HTLV1-transformed T-cells. -
细胞定位
Secreted. - Information by UniProt
图片
实验方案
To our knowledge, customised protocols are not required for this product. Please try the standard protocols listed below and let us know how you get on.
数据表及文件
-
SDS download
-
Datasheet download
文献 (0)
ab134632 尚未被引用在任何文献中。