Acetylcholine chloride，Endogenous neurotransmitter (ab120469)
Key features and details
- Endogenous neurotransmitter active at acetylcholine receptors
- CAS Number: 60-31-1
- Soluble in water to 100 mM, in DMSO to 100 mM and in ethanol to 75 mM
- Form / State: Solid
- Source: Synthetic
概述
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产品名称
Acetylcholine chloride，Endogenous neurotransmitter -
描述
Endogenous neurotransmitter active at acetylcholine receptors -
别名
- ACh
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生物学描述
Endogenous neurotransmitter in both the central and peripheral nervous systems. Displays activity at the nicotinic and muscarinic acetylcholine receptors. Also implicated in numerous non-neuronal roles.
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常规说明
Please note this compound is extremely hygroscopic. Exposure to moist air should be avoided.
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CAS编号
60-31-1 -
化学结构
性能
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化学名称
2-(Acetyloxy)-N,N,N-trimethylethanaminium chloride -
分子量
181.66 -
分子式
C7H16ClNO2 -
存放说明
Store at Room Temperature. Store under desiccating conditions. The product can be stored for up to 12 months. -
溶解度概述
Soluble in water to 100 mM, in DMSO to 100 mM and in ethanol to 75 mM -
处理
Wherever possible, you should prepare and use solutions on the same day. However, if you need to make up stock solutions in advance, we recommend that you store the solution as aliquots in tightly sealed vials at -20°C. Generally, these will be useable for up to one month. Before use, and prior to opening the vial we recommend that you allow your product to equilibrate to room temperature for at least 1 hour.
Refer to SDS for further information
Need more advice on solubility, usage and handling? Please visit our frequently asked questions (FAQ) page for more details.
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SMILES
Cl.[CH2-][N+](C)(C)CCOC(=O)C -
来源
Synthetic
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研究领域
图片
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Chemical Structure - Acetylcholine chloride, Endogenous neurotransmitter (ab120469)2D chemical structure image of ab120469, Acetylcholine chloride, Endogenous neurotransmitter
实验方案
To our knowledge, customised protocols are not required for this product. Please try the standard protocols listed below and let us know how you get on.
文献 (1)
ab120469 被引用在 1 文献中.
- Radzicki D et al. Loss of M1 Receptor Dependent Cholinergic Excitation Contributes to mPFC Deactivation in Neuropathic Pain. J Neurosci 37:2292-2304 (2017). PubMed: 28137966