概述

  • 产品名称Anti-SGK3抗体
    参阅全部 SGK3 一抗
  • 描述
    兔多克隆抗体to SGK3
  • 经测试应用适用于: IHC-Pmore details
  • 种属反应性
    与反应: Human
    预测可用于: Mouse不与反应: Rat
  • 免疫原

    Synthetic peptide corresponding to Human SGK3 (C terminal) conjugated to Keyhole Limpet Haemocyanin (KLH).

  • 阳性对照
    • Kidney

性能

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应用

Our Abpromise guarantee covers the use of ab61819 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
IHC-P Use a concentration of 5 µg/ml.

靶标

  • 功能Involved in the activation of potassium channels. Mediates cell IL-3-dependent survival signals.
  • 组织特异性Expressed in most tissues with highest levels in pancreas, kidney liver, heart and brain and lower levels in lung, placenta and skeletal muscle.
  • 序列相似性Belongs to the protein kinase superfamily. AGC Ser/Thr protein kinase family.
    Contains 1 AGC-kinase C-terminal domain.
    Contains 1 protein kinase domain.
    Contains 1 PX (phox homology) domain.
  • 细胞定位Cytoplasmic vesicle. Early endosome. Localized in vesicle-like structures and in the early endosome.
  • Information by UniProt
  • 数据库链接
  • 别名
    • CISK antibody
    • Cytokine independent survival kinase antibody
    • DKFZp781N0293 antibody
    • Serine/threonine protein kinase Sgk3 antibody
    • Serine/threonine-protein kinase Sgk3 antibody
    • Serum/glucocorticoid regulated kinase 3 antibody
    • Serum/glucocorticoid regulated kinase family member 3 antibody
    • Serum/glucocorticoid regulated kinase like antibody
    • Serum/glucocorticoid-regulated kinase 3 antibody
    • Serum/glucocorticoid-regulated kinase-like antibody
    • SGK 2 antibody
    • SGK 3 antibody
    • SGK2 antibody
    • Sgk3 antibody
    • SGK3_HUMAN antibody
    • SGKL antibody
    see all

Anti-SGK3 antibody 图像

  • ab61819, at 5µg/ml, staining human SGK3 in kidney by immunohistochemistry. Formalin-fixed paraffin-embedded tissue.

Anti-SGK3 antibody (ab61819)参考文献

ab61819 has not yet been referenced specifically in any publications.

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