Anti-Sarcomeric Alpha Actinin抗体(ab72592)

ab72592 被引用在 20 文献中.

• Veltrop M  et al. A dystrophic Duchenne mouse model for testing human antisense oligonucleotides. PLoS One 13:e0193289 (2018). PubMed: 29466448
• Jirka SMG  et al. Cyclic Peptides to Improve Delivery and Exon Skipping of Antisense Oligonucleotides in a Mouse Model for Duchenne Muscular Dystrophy. Mol Ther 26:132-147 (2018). WB ; Mouse . PubMed: 29103911
• Giacomazzi G  et al. MicroRNAs promote skeletal muscle differentiation of mesodermal iPSC-derived progenitors. Nat Commun 8:1249 (2017). PubMed: 29093487
• Brinegar AE  et al. Extensive alternative splicing transitions during postnatal skeletal muscle development are required for calcium handling functions. Elife 6:N/A (2017). PubMed: 28826478
• Giudice J  et al. Neonatal cardiac dysfunction and transcriptome changes caused by the absence of Celf1. Sci Rep 6:35550 (2016). WB ; Mouse . PubMed: 27759042
• Quijada P  et al. Nuclear Calcium/Calmodulin-dependent Protein Kinase II Signaling Enhances Cardiac Progenitor Cell Survival and Cardiac Lineage Commitment. J Biol Chem 290:25411-26 (2015). WB ; Mouse . PubMed: 26324717
• Winter L  et al. Plectin isoform P1b and P1d deficiencies differentially affect mitochondrial morphology and function in skeletal muscle. Hum Mol Genet 24:4530-44 (2015). PubMed: 26019234
• Quattrocelli M  et al. Mesodermal iPSC-derived progenitor cells functionally regenerate cardiac and skeletal muscle. J Clin Invest 125:4463-82 (2015). WB ; Mouse . PubMed: 26571398
• Verhaart IE  et al. The Dynamics of Compound, Transcript, and Protein Effects After Treatment With 2OMePS Antisense Oligonucleotides in mdx Mice. Mol Ther Nucleic Acids 3:e148 (2014). WB . PubMed: 24549299
• Giudice J  et al. Alternative splicing regulates vesicular trafficking genes in cardiomyocytes during postnatal heart development. Nat Commun 5:3603 (2014). WB ; Mouse . PubMed: 24752171
• Turnbull IC  et al. Advancing functional engineered cardiac tissues toward a preclinical model of human myocardium. FASEB J 28:644-54 (2014). IHC-P, IF . PubMed: 24174427
• van Putten M  et al. Low dystrophin levels increase survival and improve muscle pathology and function in dystrophin/utrophin double-knockout mice. FASEB J 27:2484-95 (2013). PubMed: 23460734
• van den Bergen JC  et al. Clinical characterisation of Becker muscular dystrophy patients predicts favourable outcome in exon-skipping therapy. J Neurol Neurosurg Psychiatry N/A:N/A (2013). Human . PubMed: 23667215
• Veltrop M  et al. Generation of embryonic stem cells and mice for duchenne research. PLoS Curr 5:N/A (2013). WB ; Mouse . PubMed: 24057032
• van den Bergen JC  et al. Dystrophin levels and clinical severity in Becker muscular dystrophy patients. J Neurol Neurosurg Psychiatry N/A:N/A (2013). Human . PubMed: 24292997
• Spitali P  et al. DMD transcript imbalance determines dystrophin levels. FASEB J 27:4909-16 (2013). PubMed: 23975932
• Tanganyika-de Winter CL  et al. Long-term Exon Skipping Studies With 2'-O-Methyl Phosphorothioate Antisense Oligonucleotides in Dystrophic Mouse Models. Mol Ther Nucleic Acids 1:e44 (2012). PubMed: 23344236
• Desroches BR  et al. Functional scaffold-free 3-D cardiac microtissues: a novel model for the investigation of heart cells. Am J Physiol Heart Circ Physiol 302:H2031-42 (2012). IHC-Fr ; Rat . PubMed: 22427522
• Verhaart IE & Aartsma-Rus A The effect of 6-thioguanine on alternative splicing and antisense-mediated exon skipping treatment for duchenne muscular dystrophy. PLoS Curr 4: (2012). WB ; Human . PubMed: 23259153
• Spitalieri P  et al. Identification of multipotent cytotrophoblast cells from human first trimester chorionic villi. Cloning Stem Cells 11:535-56 (2009). IHC-P ; Mouse . PubMed: 20025524