Anti-Raet1c抗体[CX1] (Phycoerythrin) (ab95800)

概述

  • 产品名称Anti-Raet1c抗体[CX1] (Phycoerythrin)
    参阅全部 Raet1c 一抗
  • 描述
    大鼠单克隆抗体[CX1] to Raet1c (Phycoerythrin)
  • 偶联物Phycoerythrin. Ex: 488nm, Em: 575nm
  • 特异性ab95800 reacts with mouse retinoic acid early inducible gamma (Raet1c ) and weakly cross-reacts with RAE-1 alpha and beta, but not with delta and epsilon.
  • 经测试应用适用于: Flow Cytmore details
  • 种属反应性
    与反应: Mouse
  • 免疫原

    CHO cells stably transfected with Mouse Raet1c.

  • 阳性对照
    • YAC-1 cell line and mouse splenocytes.

性能

  • 形式Liquid
  • 存放说明Shipped at 4°C. Store at +4°C.
  • 存储溶液Preservative: 0.09% Sodium Azide
  • Concentration information loading...
  • 纯度Protein G purified
  • 克隆单克隆
  • 克隆编号CX1
  • 同种型IgG2b
  • 轻链类型kappa
  • 研究领域

应用

Our Abpromise guarantee covers the use of ab95800 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
Flow Cyt Use 0.06-0.125µg for 105-8 cells. Perform staining in a final volume of 100 µl.

靶标

  • 相关性Retinoic acid early-inducible protein 1-gamma (Raet1c) belongs to the RAE-1 family of NKG2D ligands. They are induced by retinoic acid and expressed predominantly in embryonic brain during early embryogenesis. Detected at high levels in 7, 11 and 14-day-old embryos but not in 18-day-old embryos. Very low levels are detected in adults, however the presence of retinoic acid or certain disease conditions (such as tumors) can induce up-regulation of this protein at the cell surface.
  • 数据库链接
  • 别名
    • RAE-1-gamma antibody
    • Retinoic acid early-inducible protein 1-gamma antibody

Anti-Raet1c antibody [CX1] (Phycoerythrin) 图像

  • Flow cytometry staining of Yac-1 cell line with 0.06 µg of Rat IgG2b ? Isotype Control PE (open histogram) or 0.06 µg of ab95800 (filled histogram). Total viable cells were used for analysis.

Anti-Raet1c antibody [CX1] (Phycoerythrin) (ab95800)参考文献

ab95800 has not yet been referenced specifically in any publications.

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