概述

  • 产品名称Anti-PEX6抗体
    参阅全部 PEX6 一抗
  • 描述
    兔多克隆抗体to PEX6
  • 经测试应用适用于: WBmore details
  • 种属反应性
    与反应: Human
  • 免疫原

    Recombinant fragment, corresponding to a region within amino acids 180-432 of Human PEX6 (NP_000278).

  • 阳性对照
    • A549 whole cell lysate; HepG2 cell lysate

性能

  • 形式Liquid
  • 存放说明Shipped at 4°C. Upon delivery aliquot and store at -20°C or -80°C. Avoid repeated freeze / thaw cycles.
  • 存储溶液Preservative: 0.01% Thimerosal (merthiolate)
    Constituents: 10% Glycerol, 0.1M Tris, 0.1M Glycine, pH 7.0
  • Concentration information loading...
  • 纯度Immunogen affinity purified
  • 克隆多克隆
  • 同种型IgG
  • 研究领域

应用

Our Abpromise guarantee covers the use of ab96330 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB 1/500 - 1/3000. Predicted molecular weight: 104 kDa.

靶标

  • 相关性This gene encodes a member of the AAA (ATPases associated with diverse cellular activities) family of ATPases. This member is a predominantly cytoplasmic protein, which plays a direct role in peroxisomal protein import and is required for PTS1 (peroxisomal targeting signal 1, a C-terminal tripeptide of the sequence ser-lys-leu) receptor activity. Mutations in this gene cause peroxisome biogenesis disorders of complementation group 4 and complementation group 6.
  • 细胞定位Cytoplasm. Peroxisome membrane. Note: Associated with peroxisomal membranes.
  • 数据库链接
  • 别名
    • PAF 2 antibody
    • PAF2 antibody
    • PBD4A antibody
    • PDB4B antibody
    • Peroxin 6 antibody
    • Peroxisomal AAA type ATPase 1 antibody
    • Peroxisomal biogenesis factor 6 antibody
    • Peroxisomal type ATPase 1 antibody
    • Peroxisome assembly factor 2 antibody
    • PXAAA1 antibody
    see all

Anti-PEX6 antibody 图像

  • Anti-PEX6 antibody (ab96330) at 1/1000 dilution + A549 whole cell lysate at 30 µg

    Predicted band size : 104 kDa

Anti-PEX6 antibody (ab96330)参考文献

ab96330 has not yet been referenced specifically in any publications.

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