概述

  • 产品名称Anti-OPA3抗体
  • 描述
    小鼠多克隆抗体to OPA3
  • 经测试应用适用于: WBmore details
  • 种属反应性
    与反应: Human
  • 免疫原

    Full length human OPA3 protein (NP_079412)

  • 阳性对照
    • OPA3 transfected 293T cell lysate.

性能

  • 形式Liquid
  • 存放说明Shipped at 4°C. Upon delivery aliquot and store at -20°C or -80°C. Avoid repeated freeze / thaw cycles.
  • 存储溶液Preservative: None
    Constituents: PBS, pH 7.2
  • Concentration information loading...
  • 纯度Protein A purified
  • 克隆多克隆
  • 同种型IgG
  • 研究领域

应用

Our Abpromise guarantee covers the use of ab69163 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB 1/500 - 1/1000. Detects a band of approximately 20 kDa (predicted molecular weight: 20 kDa).

靶标

  • 相关性OPA3 may play some role in mitochondrial processes. Defects in OPA3 are the cause of 3-methylglutaconic aciduria type 3 (MGA3), also known as optic atrophy plus syndrome, and of optic atrophy type 3 (OPA3), also known as autosomal dominant optic atrophy and cataract (ADOAC).
  • 细胞定位Mitochondrial
  • 数据库链接
  • 别名
    • FLJ22187 antibody
    • FLJ25932 antibody
    • Iraqi Jewish optic atrophy plus (3 methylglutaconicaciduria type 3) antibody
    • MGA3 antibody
    • MGC75494 antibody
    • OPA 3 antibody
    • OPA3 protein antibody
    • Optic atrophy 3 (autosomal recessive, with chorea and spastic paraplegia) antibody
    • Optic atrophy 3 (Iraqi Jewish 'optic atrophy plus') antibody
    • Optic atrophy 3 antibody
    • Optic atrophy 3 protein antibody
    see all

Anti-OPA3 antibody 图像

  • All lanes : Anti-OPA3 antibody (ab69163) at 1/500 dilution

    Lane 1 : OPA3 transfected 293T cell lysate
    Lane 2 : Non-transfected 293T cell lysate

    Lysates/proteins at 25 µg per lane.

    Secondary
    Goat Anti-Mouse IgG (H&L)-HRP Conjugate at 1/2500 dilution

    Predicted band size : 20 kDa
    Observed band size : 20 kDa

Anti-OPA3 antibody (ab69163)参考文献

ab69163 has not yet been referenced specifically in any publications.

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