概述

  • 产品名称Anti-MICAL1抗体
    参阅全部 MICAL1 一抗
  • 描述
    兔多克隆抗体to MICAL1
  • 经测试应用适用于: WBmore details
  • 种属反应性
    与反应: Human
    预测可用于: Rat, Rabbit, Horse, Cow, Cat, Pig
  • 免疫原

    Synthetic peptide corresponding to a region within C terminal amino acids 920-968 (WQLDQELRGY MNREENLKTA ADRQAEDQVL RKLVDLVNQR DALIRFQEER) of Human MICAL1 (NP_001152763; UniProt Q8TDZ2 isoform 2).

  • 阳性对照
    • Human placenta lysate

性能

应用

Our Abpromise guarantee covers the use of ab116458 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB Use a concentration of 1 µg/ml. Predicted molecular weight: 109 kDa. Good results were obtained when blocked with 5% non-fat dry milk in 0.05% PBS-T.

靶标

  • 功能May be a cytoskeletal regulator that connects NEDD9 to intermediate filaments.
  • 组织特异性Expressed in the thymus, lung, spleen, kidney, testis and hematopoietic cells.
  • 序列相似性Contains 1 CH (calponin-homology) domain.
    Contains 1 LIM zinc-binding domain.
  • 结构域The C-terminal coiled coil part contains the plexin-interacting region.
  • 细胞定位Cytoplasm. Cytoplasm > cytoskeleton.
  • Information by UniProt
  • 数据库链接
  • 别名
    • CasL interacting molecule antibody
    • DKFZp434B1517 antibody
    • FLJ11937 antibody
    • FLJ21739 antibody
    • MICA1_HUMAN antibody
    • MICAL 1 antibody
    • MICAL antibody
    • MICAL1 antibody
    • microtubule associated monoxygenase, calponin and LIM domain containing 1 antibody
    • Molecule interacting with CasL protein 1 antibody
    • NEDD9 interacting protein with calponin homology and LIM domains antibody
    • NEDD9-interacting protein with calponin homology and LIM domains antibody
    • NICAL antibody
    • OTTHUMP00000016969 antibody
    see all

Anti-MICAL1 antibody 图像

  • Anti-MICAL1 antibody (ab116458) at 1 µg/ml + Human placenta lysate at 10 µg

    Predicted band size : 109 kDa

Anti-MICAL1 antibody (ab116458)参考文献

ab116458 has not yet been referenced specifically in any publications.

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