人Thromboxane synthase肽(ab72365)

概述

描述

  • 性质Synthetic
  • 氨基酸序列
    • 种属Human
    • 序列TNPDCQEKLLREVDVFKEK
    • 氨基酸359 to 377

相关产品

技术指标

Our Abpromise guarantee covers the use of ab72365 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

  • 应用

    Blocking - Blocking peptide for Anti-Thromboxane synthase antibody (ab39362)

  • 形式Liquid
  • Concentration information loading...

制备和贮存

  • 稳定性和存储

    Shipped at 4°C. Upon delivery aliquot and store at -20°C. Avoid freeze / thaw cycles.

    Preservative: None
    Constituents: PBS

常规信息

  • 别名
    • BDPLT14
    • CYP5
    • CYP5A1
    • Cytochrome P450 5A1
    • Cytochrome P450, family 5, subfamily A, polypeptide 1
    • FLJ52771
    • GHOSAL
    • Platelet cytochrome P450 subfamily V
    • Tbxas1
    • THAS
    • THAS_HUMAN
    • Thromboxane A synthase 1
    • Thromboxane A synthase 1 (platelet)
    • Thromboxane A synthase 1 (platelet, cytochrome P450, family 5, subfamily A)
    • Thromboxane A synthase 1 platelet cytochrome P450 subfamily V
    • Thromboxane-A synthase
    • TS
    • TXA synthase
    • TXAS
    • TXS
    see all
  • 组织特异性Platelets, lung, kidney, spleen, macrophages and lung fibroblasts.
  • 疾病相关Defects in TBXAS1 are the cause of Ghosal hematodiaphyseal dysplasia (GHDD) [MIM:231095]. GHDD is a rare autosomal recessive disorder characterized by increased bone density with predominant diaphyseal involvement and aregenerative corticosteroid-sensitive anemia. Aregenerative anemia is characterized by bone marrow failure, so that functional marrow cells are regenerated slowly or not at all.
    Defects in TBXAS1 are the cause of thromboxane synthetase deficiency (TBXAS1 deficiency) [MIM:274180]. It is characterized by hemorrhagic diathesis.
  • 序列相似性Belongs to the cytochrome P450 family.
  • 细胞定位Endoplasmic reticulum membrane.
  • Information by UniProt

Human Thromboxane synthase peptide (ab72365)参考文献

ab72365 has not yet been referenced specifically in any publications.

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