Anti-Glycogen synthase 1抗体(ab123377)

概述

  • 产品名称
    Anti-Glycogen synthase 1抗体
    参阅全部 Glycogen synthase 1 一抗
  • 描述
    兔多克隆抗体to Glycogen synthase 1
  • 经测试应用
    适用于: WBmore details
  • 种属反应性
    与反应: Mouse, Rat, Human
  • 免疫原

    Synthetic peptide:

    PSPSL

    , corresponding to a region around amino acids 644-648 of Human Glycogen synthase 1 (P13807 ).

  • 阳性对照
    • HeLa and 293 cell extracts

性能

应用

Our Abpromise guarantee covers the use of ab123377 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB 1/500. Detects a band of approximately 84 kDa (predicted molecular weight: 84 kDa). Incubate membrane with diluted antibody in 5% nonfat milk, 1X TBS, 0.1% Tween-20 at 4°C with gentle shaking over night.

靶标

  • 功能
    Transfers the glycosyl residue from UDP-Glc to the non-reducing end of alpha-1,4-glucan.
  • 通路
    Glycan biosynthesis; glycogen biosynthesis.
  • 疾病相关
    Defects in GYS1 are the cause of muscle glycogen storage disease type 0 (GSD0b) [MIM:611556]; also known as muscle glycogen synthase deficiency. GSD0b is a metabolic disorder characterized by fasting hypoglycemia presenting in infancy or early childhood. The role of muscle glycogen is to provide critical energy during bursts of activity and sustained muscle work.
  • 序列相似性
    Belongs to the glycosyltransferase 3 family.
  • Information by UniProt
  • 数据库链接
  • 别名
    • Glycogen [starch] synthase antibody
    • Glycogen synthase 1 (muscle) antibody
    • Glycogen synthase 1 antibody
    • GSY antibody
    • GYS antibody
    • Gys1 antibody
    • GYS1_HUMAN antibody
    • muscle antibody
    see all

Anti-Glycogen synthase 1 antibody 图像

  • All lanes : Anti-Glycogen synthase 1 antibody (ab123377) at 1/500 dilution

    Lane 1 : HeLa cell extract
    Lane 2 : 293 cell extract


    Predicted band size : 84 kDa

实验方案

Anti-Glycogen synthase 1 antibody (ab123377)参考文献

ab123377 has not yet been referenced specifically in any publications.

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