RabMAb

Anti-GCN2 (phospho T899)抗体[EPR2320Y] (ab75836)

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ab75836 被引用在 8 文献中.

  • Chaveroux C  et al. In vivo imaging of the spatiotemporal activity of the eIF2a-ATF4 signaling pathway: Insights into stress and related disorders. Sci Signal 8:rs5 (2015). WB ; Mouse . PubMed: 25921292
  • Wengrod J  et al. Phosphorylation of eIF2a triggered by mTORC1 inhibition and PP6C activation is required for autophagy and is aberrant in PP6C-mutated melanoma. Sci Signal 8:ra27 (2015). WB ; Human . PubMed: 25759478
  • Roffé M  et al. IMPACT is a developmentally regulated protein in neurons that opposes the eukaryotic initiation factor 2a kinase GCN2 in the modulation of neurite outgrowth. J Biol Chem 288:10860-9 (2013). WB ; Mouse . PubMed: 23447528
  • Tattoli I  et al. Listeria phospholipases subvert host autophagic defenses by stalling pre-autophagosomal structures. EMBO J 32:3066-78 (2013). PubMed: 24162724
  • Morotomi-Yano K  et al. Nanosecond pulsed electric fields act as a novel cellular stress that induces translational suppression accompanied by eIF2a phosphorylation and 4E-BP1 dephosphorylation. Exp Cell Res 318:1733-44 (2012). WB . PubMed: 22652449
  • Anderson BR  et al. Nucleofection induces transient eIF2a phosphorylation by GCN2 and PERK. Gene Ther : (2012). WB . PubMed: 22301437
  • Tattoli I  et al. The bacterial and cellular determinants controlling the recruitment of mTOR to the Salmonella-containing vacuole. Biol Open 1:1215-25 (2012). WB . PubMed: 23259056
  • Cai Q & Brooks HL Phosphorylation of eIF2a via the general control kinase, GCN2, modulates the ability of renal medullary cells to survive high urea stress. Am J Physiol Renal Physiol 301:F1202-7 (2011). WB ; Mouse . PubMed: 21880833

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