概述

  • 产品名称Anti-EXTL1抗体
    参阅全部 EXTL1 一抗
  • 描述
    兔多克隆抗体to EXTL1
  • 经测试应用适用于: ICC/IF, IHC-Pmore details
  • 种属反应性
    与反应: Human
  • 免疫原

    Recombinant fragment: ALPPRPRPGA SQGWPRWLDA ELLQSFSQPG ELPEDAVSPP QAPHGGSCNW ESCFDTSKCR GDGLKVFVYP AVGTISETHR RILASIEGS, corresponding to amino acids 33-121 of Human EXTL1 (Q92935).

  • 阳性对照
    • Human thyroid gland tissue

性能

应用

Our Abpromise guarantee covers the use of ab122410 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
ICC/IF Use a concentration of 1 - 4 µg/ml.

Recommend PFA Fixation and Triton X-100 treatment

IHC-P 1/20 - 1/50. Perform heat mediated antigen retrieval with citrate buffer pH 6 before commencing with IHC staining protocol.

靶标

  • 功能Probable glycosyltransferase.
  • 通路Protein modification; protein glycosylation.
  • 序列相似性Belongs to the glycosyltransferase 47 family.
  • 细胞定位Endoplasmic reticulum membrane.
  • Information by UniProt
  • 数据库链接
  • 别名
    • Alpha N acetylglucosaminyltransferase II antibody
    • Exostoses (multiple) like 1 antibody
    • Exostosin like glycosyltransferase 1 antibody
    • Exostosin-L antibody
    • Exostosin-like 1 antibody
    • EXTL antibody
    • Extl1 antibody
    • EXTL1_HUMAN antibody
    • Glucuronosyl-N-acetylglucosaminyl-proteoglycan 4-alpha-N-acetylglucosaminyltransferase antibody
    • Glucuronyl N acetylglucosaminylproteoglycan alpha 1,4 N acetylglucosaminyltransferase antibody
    • MGC70794 antibody
    • Multiple exostosis-like protein antibody
    see all

Anti-EXTL1 antibody 图像

  • Immunofluorescent staining of Human cell line U-2 OS shows positivity in nucleus but not nucleoli and vesicles. Recommended concentration of ab122410 1-4 µg/ml. Cells treated with PFA/Triton X-100.
  • ab122410 at 1/20 dilution staining EXTL1 in paraffin-embedded Human thyroid gland tissue by Immunohistochemistry.

Anti-EXTL1 antibody (ab122410)参考文献

ab122410 has not yet been referenced specifically in any publications.

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