概述

  • 产品名称Anti-ERGI3抗体
    参阅全部 ERGI3 一抗
  • 描述
    兔多克隆抗体to ERGI3
  • 经测试应用适用于: WB, ELISA, ICC/IFmore details
  • 种属反应性
    与反应: Mouse, Human
  • 免疫原

    Synthetic peptide derived from an internal sequence of human ERGI3.

  • 阳性对照
    • HepG2 cells; LOVO cells; NIH-3T3 cells.

性能

  • 形式Liquid
  • 存放说明Shipped at 4°C. Store at -20°C. Stable for 12 months at -20°C.
  • 存储溶液Preservative: 0.02% Sodium Azide
    Constituents: 50% Glycerol, PBS (without Mg2+ and Ca2+), 150mM Sodium chloride, pH 7.4
  • Concentration information loading...
  • 纯度Immunogen affinity purified
  • 克隆多克隆
  • 同种型IgG
  • 研究领域

应用

Our Abpromise guarantee covers the use of ab64902 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB 1/500 - 1/1000. Detects a band of approximately 43 kDa (predicted molecular weight: 43 kDa).
ELISA 1/20000.
ICC/IF 1/500 - 1/1000.

靶标

  • 功能Possible role in transport between endoplasmic reticulum and Golgi.
  • 序列相似性Belongs to the ERGIC family.
  • 细胞定位Endoplasmic reticulum-Golgi intermediate compartment membrane. Golgi apparatus > cis-Golgi network membrane. Endoplasmic reticulum membrane. Cycles between the endoplasmic reticulum and the Golgi.
  • Information by UniProt
  • 数据库链接
  • 别名
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    • ERGI3_HUMAN antibody
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    • ergic3 antibody
    • ERV46 antibody
    • NY BR 84 antibody
    • PRO0989 antibody
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    • Serologically defined breast cancer antigen NY-BR-84 antibody
    see all

Anti-ERGI3 antibody 图像

  • All lanes : Anti-ERGI3 antibody (ab64902) at 1/500 dilution

    Lane 1 : extracts from LOVO cells
    Lane 2 : extracts from NIH-3T3 cells
    Lane 3 : NIH-3T3 cells with immunizing peptide at 5 µg

    Lysates/proteins at 10 µg per lane.


    Predicted band size : 43 kDa
    Observed band size : 43 kDa
  • ab64902 at 1/500 dilution staining HepG2 cells without (left) and with (right) immunizing peptide.

Anti-ERGI3 antibody (ab64902)参考文献

ab64902 has not yet been referenced specifically in any publications.

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