Anti-Calneuron 1抗体(ab117584)

概述

  • 产品名称Anti-Calneuron 1抗体
    参阅全部 Calneuron 1 一抗
  • 描述
    兔多克隆抗体to Calneuron 1
  • 经测试应用适用于: WB, IHC-Pmore details
  • 种属反应性
    与反应: Human
    预测可用于: Mouse, Rat
  • 免疫原

    Synthetic peptide conjugated to KLH from the N-terminus of Human Calneuron 1

  • 阳性对照
    • Human brain cortex tissue

性能

  • 形式Liquid
  • 存放说明Shipped at 4°C. Store at +4°C short term (1-2 weeks). Store at -20°C or -80°C. Avoid freeze / thaw cycle.
  • 存储溶液Preservative: 0.02% Sodium azide
    Constituent: 99% PBS
  • Concentration information loading...
  • 纯度Immunogen affinity purified
  • 克隆多克隆
  • 同种型IgG
  • 研究领域

应用

Our Abpromise guarantee covers the use of ab117584 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

应用 Ab评论 说明
WB Use a concentration of 1 - 2 µg/ml. Predicted molecular weight: 24 kDa.
IHC-P Use a concentration of 2.5 µg/ml.

靶标

  • 功能Negatively regulates Golgi-to-plasma membrane trafficking by interacting with PI4KB and inhibiting its activity (By similarity). May play a role in the physiology of neurons and is potentially important in memory and learning.
  • 组织特异性Brain specific.
  • 序列相似性Contains 2 EF-hand domains.
  • 结构域The C-terminal transmembrane domain (TMD) is necessary and sufficient for membrane targeting.
  • 细胞定位Golgi apparatus, trans-Golgi network membrane. Cytoplasm, perinuclear region. Cell membrane.
  • Information by UniProt
  • 数据库链接
  • 别名
    • CaBP8 antibody
    • CABP8_HUMAN antibody
    • Calcium binding protein 8 antibody
    • Calcium binding protein antibody
    • Calcium binding protein CABP8 antibody
    • Calcium-binding protein 8 antibody
    • Caln1 antibody
    • Calneuron I antibody
    • Calneuron-1 antibody
    see all

Anti-Calneuron 1 antibody 图像

  • Formalin-fixed and paraffin-embedded Human brain cortex tissue labelled with ab117584 at 2.5 µg/ml.

Anti-Calneuron 1 antibody (ab117584)参考文献

ab117584 has not yet been referenced specifically in any publications.

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