Anti-Blooms Syndrome蛋白Blm抗体(ab2179)

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ab2179 被引用在 18 文献中.

  • Gong Y  et al. PHF11 promotes DSB resection, ATR signaling, and HR. Genes Dev 31:46-58 (2017). WB ; Mouse . PubMed: 28115467
  • Biehs R  et al. DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination. Mol Cell 65:671-684.e5 (2017). PubMed: 28132842
  • Bou Samra E  et al. A role for Tau protein in maintaining ribosomal DNA stability and cytidine deaminase-deficient cell survival. Nat Commun 8:693 (2017). PubMed: 28947735
  • Kibe T  et al. TPP1 Blocks an ATR-Mediated Resection Mechanism at Telomeres. Mol Cell 61:236-46 (2016). PubMed: 26778124
  • Gemble S  et al. A balanced pyrimidine pool is required for optimal Chk1 activation to prevent ultrafine anaphase bridge formation. J Cell Sci 129:3167-77 (2016). WB . PubMed: 27383768
  • Wu Y  et al. EEPD1 Rescues Stressed Replication Forks and Maintains Genome Stability by Promoting End Resection and Homologous Recombination Repair. PLoS Genet 11:e1005675 (2015). WB . PubMed: 26684013
  • Gemble S  et al. Pyrimidine Pool Disequilibrium Induced by a Cytidine Deaminase Deficiency Inhibits PARP-1 Activity, Leading to the Under Replication of DNA. PLoS Genet 11:e1005384 (2015). WB . PubMed: 26181065
  • Mackay DR & Ullman KS ATR and a Chk1-Aurora B pathway coordinate postmitotic genome surveillance with cytokinetic abscission. Mol Biol Cell 26:2217-26 (2015). IF . PubMed: 25904336
  • Kumari A  et al. BLM protein mitigates formaldehyde-induced genomic instability. DNA Repair (Amst) 28:73-82 (2015). PubMed: 25770783
  • Lu H  et al. Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice. Cell Death Dis 5:e1226 (2014). WB ; Human . PubMed: 24832598
  • Petsalaki E  et al. Chk1 protects against chromatin bridges by constitutively phosphorylating BLM serine 502 to inhibit BLM degradation. J Cell Sci 127:3902-8 (2014). PubMed: 25015292
  • Zimmermann M  et al. TRF1 negotiates TTAGGG repeat-associated replication problems by recruiting the BLM helicase and the TPP1/POT1 repressor of ATR signaling. Genes Dev 28:2477-91 (2014). PubMed: 25344324
  • Hu L  et al. Replication stress and mitotic dysfunction in cells expressing simian virus 40 large T antigen. J Virol 87:13179-92 (2013). PubMed: 24067972
  • Schrötter A  et al. FE65 regulates and interacts with the Bloom syndrome protein in dynamic nuclear spheres - potential relevance to Alzheimer's disease. J Cell Sci 126:2480-92 (2013). WB ; Human . PubMed: 23572515
  • Grabarz A  et al. A role for BLM in double-strand break repair pathway choice: prevention of CtIP/Mre11-mediated alternative nonhomologous end-joining. Cell Rep 5:21-8 (2013). PubMed: 24095737
  • Bosco N & de Lange T A TRF1-controlled common fragile site containing interstitial telomeric sequences. Chromosoma 121:465-74 (2012). PubMed: 22790221
  • Singh DK  et al. The human RecQ helicases BLM and RECQL4 cooperate to preserve genome stability. Nucleic Acids Res : (2012). WB ; Human . PubMed: 22544709
  • Ke Y  et al. PICH and BLM limit histone association with anaphase centromeric DNA threads and promote their resolution. EMBO J 30:3309-21 (2011). WB, ICC/IF ; Human . PubMed: 21743438